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Posted by fangburma93 (#6) 28 minutes ago (Editorial)
The patient has been investigated and diagnosed because getting tumoral calcinosis. The particular metastatic calcification occurring in the individual has been probably related to higher calcium?��?phosphate merchandise with coexistent supplementary hyperparathyroidism perhaps aggravated simply by vitamin and mineral Deborah treatments. The person has been addressed with revulsion associated wit
Require further optimization. In addition to viral vectors, recombinant bacterial carriersRequire further optimization. In addition to viral vectors, recombinant bacterial carriers, derived from lactic acid bacteria, Salmonella and L. monocytogenes strains, have been used extensively as delivery systems being able to stimulate both systemic [29,30] and mucosal immune responses .GENE DELIVERY
Require further optimization. In addition to viral vectors, recombinant bacterial carriersRequire further optimization. In addition to viral vectors, recombinant bacterial carriers, derived from lactic acid bacteria, Salmonella and L. monocytogenes strains, have been used extensively as delivery systems being able to stimulate both systemic [29,30] and mucosal immune responses .GENE DELIVERY
Substantia nigra (SN) colocalized with TH in neurons, GFAP in astrocytes and OX-6 and OX-42 in activated microglia [166?68]. Using different carboxy terminal directed AT1 and AT2 antibodies for Western blotting, it was shown that estrogen treatment of ovariectomized rats, which was protective against 6-hydroxydopamine induced neurotoxicity in the SN, decreased AT1 and increased AT2 expression in
Substantia nigra (SN) colocalized with TH in neurons, GFAP in astrocytes and OX-6 and OX-42 in activated microglia [166?68]. Using different carboxy terminal directed AT1 and AT2 antibodies for Western blotting, it was shown that estrogen treatment of ovariectomized rats, which was protective against 6-hydroxydopamine induced neurotoxicity in the SN, decreased AT1 and increased AT2 expression in
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Exercise performance in patients with peripheral arterial disease (PAD); however, the mechanism of action is unclear. Purpose: We sought to use CMR to study the relationship between LDL reduction and superficial femoral artery (SFA) plaque burden in patients with PAD treated with lipid lowering therapy over the course of 1 year. Methods: 63 patients with mild-to-moderate symptomatic PAD (mean age
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